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Healthy heart, happy life
This editorial is published on the occasion of World Heart Day - September 28, 2014.
* For correspondence: jianghong58@gmail.com
This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
This article was originally published by Medknow Publications & Media Pvt Ltd and was migrated to Scientific Scholar after the change of Publisher.
Cardiovascular diseases have high rates of morbidity, mortality, and disability, and are the leading causes of human death irrespective of age, race, and region. According to the World Heart Federation, one third of adults over the age of 25 suffer from cardiovascular diseases globally1. Every year over 17.5 million people die from cardiovascular diseases worldwide which contributes to 30 per cent of the annual death2. The prognosis of cardiovascular diseases has been greatly improved in recent years with the development of medical technologies leading to a decrease in the morbidity and mortality. However, the morbidity and mortality due to heart diseases in the developing countries is increasing mainly due to environmental factors and unhealthy living habits. A great deal of attention is required to lead a healthy lifestyle and create an unpolluted environment to benefit the life and health of our hearts.
Smoking cessation: Smoking is closely associated with various cardiovascular diseases including coronary heart disease and hypertension3. Compared with non-smokers, the risk of cardiovascular diseases for smokers increases 1.6 fold. A report from the Asia Pacific Cohort Studies Collaboration indicates that smokers are 27 per cent more likely to develop ischaemic heart disease, 9 per cent more likely for haemorrhagic stroke, 4.5 times more likely for hypertension, and 16 times more likely for hyperlipidaemia4. Studies suggest that the risk of myocardial infarction of non-smokers who live with smokers increases by 23 per cent5. Summary analysis shows that in a person who has no history of cardiovascular disease, smoking cessation reduced the mortality of cardiovascular disease by about 2-35 per cent, which is similar to the effect antihypertensive intervention. Cessation before the age of 40 years reduces the risk of death associated with continued smoking by about 90 per cent6. For those who have heart disease, smoking cessation can reduce the overall mortality by 12-35 per cent. This effect is significantly better than antihypertensive and lipid-lowering treatments7.
Diet: Diet is crucial in the development and prevention of cardiovascular disease and is one of the key factors that one can change for a healthy heart. Abnormal blood lipid levels have been shown to have a strong correlation with the risk of coronary heart disease8, and the abnormal blood lipid levels are directly related to the diet. A diet rich in saturated fats often causes high serum cholesterol levels. Unsaturated fats, like those found in fish, nuts, seeds and vegetables, are beneficial for heart. These sources of unsaturated fats contain essential fatty acids, including omega-3 and omega-6, which are beneficial to the heart and cannot be produced by the body.
Hypertension is another important risk factor for cardiovascular diseases, which is partly attributed to a high sodium diet. The daily intake of salt should be less than 5g according to the recommendations of the WHO9.
The unhealthy diets including fast food increase the risk of hypercholesterolaemia, hypertension, and diabetes, which ultimately damage the heart. To keep a healthy heart, it is necessary to have a diet low in saturated fats and salt, but with plenty of fresh fruit and vegetables.
Exercise: Exercise can influence a variety of cardiovascular regulatory peptides, and can lower C-reactive protein (CRP) levels and delay the development of cardiovascular diseases101112. Exercise can reduce body fat levels and improve insulin sensitivity1314. Sticking to a long-term exercise regimen is an effective method to reduce CRP and both help prevent cardiovascular disease1415. Milani and colleagues16 compared the effects of cardiovascular rehabilitation training for three months in 235 coronary heart disease patients with 42 patients with no rehabilitation therapy. The results showed that the body fat index, motility, and other cardiovascular risk factors were significantly improved in the rehabilitated group16.
Pollution Control: It is suggested that environmental pollution is a greater cause to congenital heart disease than genetic factors. The development of the foetus is also influenced by a polluted environment during pregnancy and can ultimately lead to congenital heart disease. Although the epidemiological evidence is limited and inconsistencies remain, recent studies have suggested that maternal exposure to air pollution may also play a role in causing congenital anomalies, particularly congenital heart diseases1718.
The impact of pollution from the environment has been studied on 45 heart disease patients in an area of Helsinki, Finland19. Volunteers in the trial dramatically reduced the amount of blood flow into the heart after breathing polluted air for just two days. Though there was no pain associated with this change in heart function, it was a hallmark of heart disease progression. Epidemiological studies corroborate the elevated risk for cardiovascular events associated with the exposure to particle pollution (PM)2.5. PM2.5 has been associated with the increased risks of myocardial infarction, stroke, arrhythmia, and heart failure exacerbation within hours to days of exposure in susceptible individuals20.
In conclusion, cardiovascular diseases are a devastating set of diseases that are best combated by preventative measures including a healthy diet and a healthy lifestyle. The government agencies and individuals around the world should work together to create societies that promote the pursuit of a healthy heart and happy life.
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